Hot Products

Hot products from BioCrick which is a professional high-purity natural products manufacturer are well known to scientists around the world because of their high purity and stability. Each product is a chemical compound or substance produced by a living organism—that is, found in nature. In the broadest sense, natural products include any substance produced by life.Natural products remain the best sources of drugs and drug leads, and this remains true today despite the fact that many pharmaceutical companies have deemphasized natural products research in favor of HTP screening of combinatorial libraries during the past 2 decades. From 1940s to date, 131 (74.8%) out of 175 small molecule anticancer drugs are natural product-based/inspired, with 85 (48.6%) being either natural products or derived therefrom. From 1981 to date, 79 (80%) out of 99 small molecule anticancer drugs are natural product-based/inspired, with 53 (53%) being either natural products or derived therefrom. Among the 20 approved small molecule New Chemical Entities (NCEs) in 2010, a half of them are natural products.

Hot products from the professional high-purity natural products manufacturer

Cat.No. Product Name
BCN5495 Methyl isodrimeninol
Standard reference
BCN4363 Methyl pseudolarate A
Standard reference
BCN4364 Methyl pseudolarate B
Standard reference
BCN3829 Methylnissolin-3-O-glucoside
(6aR,11aR)-9,10-Dimethoxypterocarpan 3-O-β-D-glucoside(Methylnissolin-3-O- glucoside) has antioxidant activity.
BCN2841 Methylophiopogonone A
Methylophiopogonone A is a natural product from Ophiopogon japonicus.
BCN3535 Methylsyringol
Methylsyringol is a natural product from Pinus yunnanensis.
BCN3126 3-hydroxymorindone
Methyltanshinonate shows antiplasmodial and antitrypanosomal activities. It may have antioxidative activity.
BCN8446 Mogrol
Mogrol has exhibited anti-cancer activities, it suppressed leukemia cell growth via inhibition of the ERK1/2 and STAT3 pathways, in particular, through the suppression of p-ERK1/2 and p-STAT3. Mogrol significantly improved LPS-induced memory impairment in mice. It also suppressed adipogenesis by reducing CREB activation in the initial stage of cell differentiation and by activating AMPK signaling in both the early and late stages of this process.