Hot Natural Products & Bioactive Compounds

BioCrick offers high-purity natural products and bioactive compounds for scientific research. Our natural product collection includes compounds derived from plants, microorganisms and other biological sources and is widely used in pharmacological, biochemical and drug discovery research.

Natural products remain an important source of bioactive molecules and drug leads. BioCrick provides researchers with high-quality natural product compounds together with catalog numbers, product information and biological activity data.

Hot Products from BioCrick

Showing 5969 - 5976 of 9359 hot products

BioCrick hot natural products and bioactive compounds
Catalog No. Product Name
BCN4186 Mussaenoside
1. Mussaenoside has anti-inflammatory action, the mechanism associated with downregulation of nuclear factor-κB.
2. Mussaenoside can inhibit the release of pro-inflammatory cytokines induced by LPS, the production of nitric oxide (NO) and prostaglandin E2, and the expression of inducible NO synthase and cyclooxygenase-2 induced by lipopolysaccharide (LPS) in the RAW264.7 murine macrophage cell line.
BCN4187 Shanzhiside methylester
Shanzhiside methylester reduces neuropathic pain by activating spinal GLP-1 receptors and subsequently stimulating microglial β-endorphin expression via the p38 MAPK signaling.
BCN4188 Coleonol B
Coleonol B a natural product from Coleus forskohlii.
BCN4189 Tetrahydroalstonine
Tetrahydroalstonine and Raubasine preferentially block the pressor responses of post-synaptic alpha-adrenergic receptor activation due to exogenous and endogenouse noradrenaline, respectively.
BCN4190 Betulalbuside A
Standard reference
BCN4193 Sendanolactone
1. Sendanolactone possesses moderate cytotoxic activity against KB cell lines.
BCN4194 7alpha-Hydroxystigmasterol
Standard reference
BCN4196 Dehydrocrenatidine
1. Dehydrocrenatidine is a JAK-specific inhibitor, can inhibit JAK-STAT3-dependent DU145 and MDA-MB-468 cell survival and induce cell apoptosis.
2. Dehydrocrenatidine inhibits JAK2-JH1 kinase activity in vitro , represses constitutively activated JAK2 and STAT3, as well as interleukin-6-, interferon-α-, and interferon-γ-stimulated JAK activity, and STAT phosphorylation, and suppresses STAT3 and STAT1 downstream gene expression.