Ylangenyl acetateCAS# 90039-63-7 |
Quality Control & MSDS
Number of papers citing our products
Chemical structure
3D structure
Cas No. | 90039-63-7 | SDF | Download SDF |
PubChem ID | 14021304 | Appearance | Oil |
Formula | C17H26O2 | M.Wt | 262.39 |
Type of Compound | Sesquiterpenoids | Storage | Desiccate at -20°C |
Solubility | Soluble in Chloroform,Dichloromethane,Ethyl Acetate,DMSO,Acetone,etc. | ||
SMILES | CC(C)C1CCC2(C3C1C2C(=CC3)COC(=O)C)C | ||
Standard InChIKey | OBDVJFXOTPVHBB-UHFFFAOYSA-N | ||
Standard InChI | InChI=1S/C17H26O2/c1-10(2)13-7-8-17(4)14-6-5-12(9-19-11(3)18)16(17)15(13)14/h5,10,13-16H,6-9H2,1-4H3 | ||
General tips | For obtaining a higher solubility , please warm the tube at 37 ℃ and shake it in the ultrasonic bath for a while.Stock solution can be stored below -20℃ for several months. We recommend that you prepare and use the solution on the same day. However, if the test schedule requires, the stock solutions can be prepared in advance, and the stock solution must be sealed and stored below -20℃. In general, the stock solution can be kept for several months. Before use, we recommend that you leave the vial at room temperature for at least an hour before opening it. |
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About Packaging | 1. The packaging of the product may be reversed during transportation, cause the high purity compounds to adhere to the neck or cap of the vial.Take the vail out of its packaging and shake gently until the compounds fall to the bottom of the vial. 2. For liquid products, please centrifuge at 500xg to gather the liquid to the bottom of the vial. 3. Try to avoid loss or contamination during the experiment. |
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Shipping Condition | Packaging according to customer requirements(5mg, 10mg, 20mg and more). Ship via FedEx, DHL, UPS, EMS or other couriers with RT, or blue ice upon request. |
Targets | PGE | NOS | COX | EGFR |
Ylangenyl acetate Dilution Calculator
Ylangenyl acetate Molarity Calculator
1 mg | 5 mg | 10 mg | 20 mg | 25 mg | |
1 mM | 3.8111 mL | 19.0556 mL | 38.1112 mL | 76.2224 mL | 95.278 mL |
5 mM | 0.7622 mL | 3.8111 mL | 7.6222 mL | 15.2445 mL | 19.0556 mL |
10 mM | 0.3811 mL | 1.9056 mL | 3.8111 mL | 7.6222 mL | 9.5278 mL |
50 mM | 0.0762 mL | 0.3811 mL | 0.7622 mL | 1.5244 mL | 1.9056 mL |
100 mM | 0.0381 mL | 0.1906 mL | 0.3811 mL | 0.7622 mL | 0.9528 mL |
* Note: If you are in the process of experiment, it's necessary to make the dilution ratios of the samples. The dilution data above is only for reference. Normally, it's can get a better solubility within lower of Concentrations. |
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Inhibitory effects of beta-chamigrenal, isolated from the fruits of Schisandra chinensis, on lipopolysaccharide-induced nitric oxide and prostaglandin E2 production in RAW 264.7 macrophages [corrected].[Pubmed:24871206]
Planta Med. 2014 Jun;80(8-9):655-61.
Much is known about the bioactive properties of lignans from the fruits of Schisandra chinensis. However, very little work has been done to determine the properties of sesquiterpenes in the fruits of S. chinensis. The aim of the present study was to investigate the anti-inflammatory potential of new sesquiterpenes (beta-chamigrenal, beta-chamigrenic acid, alpha-ylangenol, and alpha-Ylangenyl acetate) isolated from the fruits of S. chinensis and to explore their effect on macrophages stimulated with lipopolysaccharide. Of these four sesquiterpenes, beta-chamigrenal most significantly suppressed lipopolysaccharide-induced nitric oxide and prostaglandin E2 production in RAW 264.7 macrophages (47.21 +/- 4.54 % and 51.61 +/- 3.95 % at 50 microM, respectively). Molecularly, the inhibitory activity of beta-chamigrenal on nitric oxide production was mediated by suppressing inducible nitric oxide synthase activity but not its expression. In the prostaglandin E2 synthesis pathway, beta-chamigrenal prevented the upregulation of inducible microsomal prostaglandin E synthase-1 expression after stimulation with lipopolysaccharide. Conversely, beta-chamigrenal had no effect on the expression and enzyme activity of cyclooxygenase-2. In addition, the expression of early growth response factor-1, a key transcription factor of microsomal prostaglandin E synthase-1 expression, was inhibited by beta-chamigrenal. These results may suggest a possible anti-inflammatory activity of beta-chamigrenal which has to be proven in in vivo experiments.